Being pedantic, but I wouldn't frame it this way. The pathologies in those conditions aren't that insulin levels are too high (barring accidental/intentional overdose), but a deficient effect.

There are feedback mechanisms that titrate secretion to response in cases of insulin resistance, therefore prolonged hyperglycaemia promotes insulin secretion.

Type 1 is typically due to a deficiency of insulin for various reasons.

You don't need to quote the articles conclusion at me.

1. This is a case report, not a comprehensive body of work/research
2. There are more likely explanations for his height
3. We don't even know what height he'll end up as they've only reported on forearm growth plates.

Look, whatever height you are, just come to accept it. There is precious little you can change about it now, and being fit/strong is only going to be a benefit in your future life.

I'm referring to the references used by the article you've linked. The source your article uses to link weight training to early growth plate closure refers to this happening in the case of traumatic injuries, rather than a global phenomenon. This is a risk in all paediatric traumas, and is why things like expanding prosthetics exist.

In regards to his parents' heights, there is always likely to be an element of reversion towards the mean, plus early/precocious puberty causes a reduced height anyway.

That looks far more like a case of early puberty, than exercise induced issues. The premature growth plate fusion mentioned in the references was due to traumatic injuries rather than a global effect. Also, lets not ignore that he's a 6ft 13 year old, which as stated is _tall_ for his age.